Publications

2019

Jayaprakash N, Nowak N, Eastwood E, Krueger N, Wang Z, Blackmore M (2019). Restoration of Direct Corticospinal Tract Communication Across Sites of Spinal Injury. 11 February 2019, BioRxiv https://doi.org/10.1101/546374

2018

Wang Z, Maunze B, Wang Y, Simpson M, Tsoulfas P, Blackmore M. Global connectivity and function of descending spinal input revealed by 3D microscopy and retrograde transduction. 19 October 2018, 1196-18; DOI: https://doi.org/10.1523/JNEUROSCI.1196-18.2018

2017

2016

2015

Venkatesh I, Mehra V, Wang Z, Calliff B and Blackmore M (2018). Developmental chromatin restriction of pro-growth gene networks acts as an epigenetic barrier to axon regeneration in cortical neurons. Dev Neurobiology. https://doi.org/10.1002/dneu.22605

Wang Z, Winsor K, Nienhaus C, Hess E, Blackmore MG (2017) Combined chondroitinase and KLF7 expression reduce net retraction of sensory and CST axons from sites of spinal injury. Neurobiol Dis 99:24–35.

 

Venkatesh I, Blackmore MG (2017) Selecting optimal combinations of transcription factors to promote axon regeneration: Why mechanisms matter. Neurosci Lett 652:64–73.

 

Callif BL, Maunze B, Krueger NL, Simpson MT, Blackmore MG (2017) The application of CRISPR technology to high content screening in primary neurons. Mol Cell Neuroscie 80:170-179.

Venkatesh I, Simpson MT, Coley DM, Blackmore MG (2016) Epigenetic profiling reveals a developmental decrease in promoter accessibility during cortical maturation in vivo. Neuroepigenetics 8:19–26.

 

Jayaprakash N, et al. (2016) Optogenetic Interrogation of Functional Synapse Formation by Corticospinal Tract Axons in the Injured Spinal Cord. J Neurosci 36(21):5877–90.

 

Simpson MT, et al. (2015) The tumor suppressor HHEX inhibits axon growth when prematurely expressed in developing central nervous system neurons. Mol Cell Neurosci 68. doi:10.1016/j.mcn.2015.08.008.

 

Wang Z, Reynolds A, Kirry A, Nienhaus C, Blackmore MG (2015) Overexpression of sox11 promotes corticospinal tract regeneration after spinal injury while interfering with functional recovery. J Neurosci 35(7):3139–3145.